14 : Mechanism of action: aspirin-platelet effects:
1.promotes platelet aggregation
2.activates thromboxane synthesis
3.both
4.neither
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Thromboxanes are responsible for the aggregation of platelets that form blood clots. Low-dose, long-term aspirin use irreversibly blocks the formation of thromboxane A2 in platelets, producing an inhibitory effect on platelet aggregation.
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