Aspirin and other nonsteroidal anti-inflammatory drugs (nsaid)inhibit inflammation by inhibiting an enzyme in the prostaglandin pathway, but by inhibiting this enzyme and this pathway they also inhibit pathways that protect the stomach from damage by stomach acid and that prevent aggregation of blood platelets to form blood clots. What can you conclude from this?
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Prostaglandins are lipid autacoids derived from arachidonic acid. They both sustain homeostatic functions and mediate pathogenic mechanisms, including the inflammatory response. They are generated from arachidonate by the action of cyclooxygenase (COX) isoenzymes and their biosynthesis is blocked by nonsteroidal anti-inflammatory drugs (NSAIDs), including those selective for inhibition of COX-2. Despite the clinical efficacy of NSAIDs, prostaglandins may function in both the promotion and resolution of inflammation.
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