Question

Curiosity
1. What is blood clotting? How and when
does it occur?
2. What is immunity? Name its types.
3. Why does the platelet count decrease in
dengue patient?
4. Why does our immune system fail
against pathogens like Trypanosoma and
Plasmodium?
5. What is the relation between immunity
and organ transplantation?
6. How do monocytes perform amoeboid
movement and phagocytosis?
7. How do
monocytes modify into
macrophages?​

Answer 1

Answer:

1. Blood clotting normally occurs when there is damage to a blood vessel. Platelets immediately begin to adhere to the cut edges of the vessel and release chemicals to attract even more platelets. A platelet plug is formed, and the external bleeding stops.
2. These specialized cells and parts of the immune system offer the body protection against disease. This protection is called immunity. Humans have three types of immunity — innate, adaptive, and passive: Innate immunity: Everyone is born with innate (or natural) immunity, a type of general protection.
3. When an infected mosquito bites a human, the dengue virus enters the bloodstream, it binds to platelets and replicates leading to multiplication of infectious virus. The infected platelet cells tend to destroy normal platelets which is one of the major causes for the drop in the platelet count in dengue fever.
4. sorry don't know
5. Rejection is caused by the immune system identifying the transplant as foreign, triggering a response that will ultimately destroy the transplanted organ or tissue. Long term survival of the transplant can be maintained by manipulating the immune system to reduce the risk of rejection.
6. Amoeboid cells use what is called a pseudopodia to move forward. ... Amoeboid cells are also able to carry-out a process known as phagocytosis, which literally means to eat or ingest other cells. The amoeboid cells accomplish phagocytosis by using pseudopodia to surround and engulf other cells.
7. Monocytes migrate into the intima guided by chemokines [6] and differentiate into macrophages. These macrophages then take up modified lipoproteins and, as they accumulate excess lipids, they form foam cells [3]. The foam cells die and release intracellular contents [7], which induce an inflammatory reaction.

Hope it helped u