Oxidative stress and vascular endothelial are recognized as important contributing factors in the pathogenesis of ad and dementia of vascular origin
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The most accepted hypothesis proposes that deposition of insoluble amyloid β protein (Aβ) fragments causes the neurodegeneration in AD [5]. Aβ is thought to contribute to the accumulation of reactive oxygen species (ROS) resulting in oxidative stress, neuronal damage and cognitive decline
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The most accepted hypothesis proposes that deposition of insoluble amyloid β protein (Aβ) fragments causes the neurodegeneration in AD [5]. Aβ is thought to contribute to the accumulation of reactive oxygen species (ROS) resulting in oxidative stress, neuronal damage and cognitive decline
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