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Reactive oxygen species generation and mitochondrial dysfunction in the apoptotic cell death of human myeloid leukemia hl-60 cells by a dietary compound withaferin a with concomitant protection by n-acetyl cysteine doi

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Reactive oxygen species generation and mitochondrial dysfunction in the apoptotic cell death of human myeloid leukemia hl-60 cells by a dietary compound withaferin a with concomitant protection by n-acetyl cysteine doi

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