Question

what is the most interesting discovery that brain mapper have made?​

Answer 1

Answer:

the map of our brain with the help of the fingerprints

Answer 2

Answer:

There was the dazzling array of crazy neurotech: paralyzed patients shopped and texted using an Android tablet with just their brain waves; BrainNet let three people collaboratively play a Tetris-like game using their thoughts; a first memory prosthesis boosted recall in humans; and brain-controlled robotic limbs could know their location in space or even add a “third arm” in able-bodied people.

There was the new lineup of exquisitely detailed brain maps that further unveiled the brain’s nooks and crannies: a digital museum, constructed with the help of a quarter million gamers, that showcases every bend and turn of neurons in the mouse’s retina, or a map in which billions of synapses in the mouse brain light up like the starry night.

It was the year that human brain organoids—“mini-brains” that loosely resemble the real thing during fetal development—grew their own blood supply, thrived for months inside mouse brains, and shocked the world by producing electrical patterns that resemble those seen in premature babies, launching a debate on their ethical use.

But that’s not all. Here are five neuroscience findings from 2018 that still blow our minds as we kick off the new year.

1. An infectious side to Alzheimer’s disease

Potential new drugs for Alzheimer’s have all ended up in its notorious graveyard of dreams. Despite best efforts, drugs that target two proteins that build up in Alzheimer’s disease—beta-amyloid and mutant tau—have consistently failed human drug trails.

This year, scientists are beginning to think outside the box, and new theories of how the disease is triggered and progresses are gaining steam.

In October, several studies presented some of the strongest evidence yet that herpes simplex virus type I (HSV1)—the annoying virus responsible for cold sores—may be a potential trigger. Scientists have known since the 1990s that HSV1 confers a large risk for Alzheimer’s in people who carry a specific variant of a gene called APOE4.

Most people get infected with HSV1 as children, and the virus then remains dormant until external cues such as stress reactivate it. The new theory suggests that repeated activation of the virus in adulthood in the brain could cause cumulative damage,