Question

According to Hardy- Weinberg's Principle. , the allele frequency of a population remains constant. How do you interpret the change of frequency of alleles in a population?

Answer 1

the change in allele frequency in population may b brought about by the following processes, which alter Hardy Weinberg Equillibrum
these include
mutation,
Gene Recombination
Genetic Drift or Swell Right effect including Bottle neck effect and founder effect
and some times , it is also affected by Migration including Emigration and Immigration
frequent migrations is called Gene flow
hope this helps u
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Answer 2

Hardy- Weinberg's Principle

Explanation:

The Hardy-Weinberg Theorem manages Mendelian hereditary qualities with regards to populaces of diploid, explicitly replicating people. Given a lot of presumptions (talked about beneath), this hypothesis expresses that which is follows.  

1. Allele frequencies in a populace won't change from age to age.  
2. If the allele frequencies in a populace with two alleles at a locus are p and q, at that point the normal genotype frequencies are p2, 2pq, and q2. This recurrence conveyance won't change from age to age once a populace is in Hardy-Weinberg harmony.  
3. Population size is boundless, which implies that hereditary float isn't causing arbitrary changes in allele frequencies because of testing mistake starting with one age then onto the next.  

Developmental Implications of the Hardy-Weinberg Theorem.  

• Population heterozygosity (the recurrence of heterozygotes) is most elevated when p = q = 0.5.  
• Rare alleles are found basically in heterozygotes, as they should be, given that q2 is a lot littler than 2pq when q is close to zero, and p2 is a lot littler than 2pq when p is almost zero.  
• Selection, transformation, movement, and hereditary float are the systems that impact changes in allele frequencies, and when at least one of these powers are acting, the populace damages Hardy-Weinberg suppositions, and advancement happens.
• The Hardy-Weinberg Theorem subsequently establishes an invalid model for the control of populace hereditary qualities, and is principal to the investigation of development.