Epinephrine is released from sympathetic postganglionic nerve ending----true or false? and what is the reasons
Answers
Answered by
0
Hey dear friend
Yes, epinephrine is released from postganglionic nerve endings
The population of sympathetic nonvasoconstrictor neurons consists of motility-regulating neurons and secretomotor neurons .These two groups of neurons are most likely further subdifferentiated with respect to various functions of the GIT. Most of the motility-regulating neurons and secretomotor neurons innervate neurons of the myenteric or submucosal plexus, respectively. Activation of these sympathetic neurons inhibits enteric neurons; this inhibition occurs mainly presynaptically by decrease of release of excitatory transmitter, but also postsynaptically. A few postganglionic sympathetic secretomotor neurons innervate the mucosa directly and a few postganglionic sympathetic motility-regulating neurons innervate the nonsphincteric smooth muscles, leading in both cases to inhibition (decrease of contraction or secretion) when activated. Sphincter muscles are directly innervated by sympathetic postganglionic fibers and contract when these fibers are excited.
Postganglionic nonvasoconstrictor neurons in the prevertebral ganglia receive synaptic input from enteric intestinofugal neurons and form extraspinal reflex circuits. These intestinofugal neurons are cholinergic and may have vasoactive intestinal peptide co-localized. In the guinea pig, collaterals of spinal peptidergic afferents containing substance P (SP) appear to influence only postganglionic neurons in prevertebral ganglia that express somatostatin (in addition to noradrenaline). Only these postganglionic neurons, which have secretomotor function, express the tachykinin neurokinin-receptor 1 for SP
So..
For location as it's made the release occurs
Yes, epinephrine is released from postganglionic nerve endings
The population of sympathetic nonvasoconstrictor neurons consists of motility-regulating neurons and secretomotor neurons .These two groups of neurons are most likely further subdifferentiated with respect to various functions of the GIT. Most of the motility-regulating neurons and secretomotor neurons innervate neurons of the myenteric or submucosal plexus, respectively. Activation of these sympathetic neurons inhibits enteric neurons; this inhibition occurs mainly presynaptically by decrease of release of excitatory transmitter, but also postsynaptically. A few postganglionic sympathetic secretomotor neurons innervate the mucosa directly and a few postganglionic sympathetic motility-regulating neurons innervate the nonsphincteric smooth muscles, leading in both cases to inhibition (decrease of contraction or secretion) when activated. Sphincter muscles are directly innervated by sympathetic postganglionic fibers and contract when these fibers are excited.
Postganglionic nonvasoconstrictor neurons in the prevertebral ganglia receive synaptic input from enteric intestinofugal neurons and form extraspinal reflex circuits. These intestinofugal neurons are cholinergic and may have vasoactive intestinal peptide co-localized. In the guinea pig, collaterals of spinal peptidergic afferents containing substance P (SP) appear to influence only postganglionic neurons in prevertebral ganglia that express somatostatin (in addition to noradrenaline). Only these postganglionic neurons, which have secretomotor function, express the tachykinin neurokinin-receptor 1 for SP
So..
For location as it's made the release occurs
Gargihazra:
thankuuu
welcome dear
Similar questions