Biology, asked by neelam6804, 1 year ago

Regulation of acid production by parasympathetic nervous system

Answers

Answered by Bond54132
22

There are three regulatory molecules that stimulate acid secretion (acetylcholine, histamine, gastrin) and one regulatory molecule that inhibits acid secretion (somatostatin). Acetylcholine is a neurotransmitter that is released by enteric neurons. Histamine is a paracrine that is released from ECL (enterochromaffin-like) cells. Gastrin is a hormone that is released by G cells, endocrine cells that are located in the gastric epithelium. Somatostatin is also secreted by endocrine cells of the gastric epithelium; it can act as either a paracrine or a hormone.

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Answered by nafibarli789
2

Answer:

The nervous system enables all the parts of the body to communicate with each other. It also responds to changes both outside and inside the body. The nervous system utilizes both electrical and chemical norms to send and receive messages.

Explanation:

The parasympathetic nervous system regulates bodily functions when a person exists at rest. Some of its activities include promoting digestion, activating metabolism, and assisting the body relax.

The parasympathetic nervous system exists as part of the body's autonomic nervous system. Its partner exists the sympathetic nervous system, which control's the body's fight or flight response. The parasympathetic nervous system regulates the body's ability to relax. It's sometimes named the "rest and digest" state.

Secretion of gastric acid exists regulated positively by the parasympathetic nervous system, by the hormone gastrin, and by the paracrine histamine. The final effector for each of these elements is the proton pump located in the parietal cell.

The parasympathetic nervous system regulates processes in the body such as digestion, repair, and relaxation. When the parasympathetic nervous system stands dominant in the body it conserves energy, slows heart rate, increases digestion, and relaxes sphincter muscles in the digestive tract.

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